Pathophysiological consequences of SARS CoV-2
Coronavirus outbreak occurred in late 2019 and called (COVID-19), caused by SARS-CoV-2 which firstly emerged in Wuhan city/China. This virus mostly attacks the respiratory system and therefore is more likely to destroy lung tissue and cause pneumonia; although lung is the target tissue but many organs of COVID 19 patients affect by virus invasion and showed numerous Physiological consequences. Angiotensin converting enzyme-2 (ACE2) receptors for the virus attachment are distributed in human's tissues including “lungs, heart, kidneys, intestines, brain and testes” that are recognized as a possible targets of COVID-19.The vital functions of these organs may impaired with different levels as a result of viral infection and replication. Patient with type A especially those formerly have been identified with cardiovascular diseases in particular increased blood pressure, are more expected to develop acute COVID-19 symptoms. Clot producing disorders with SARS CoV-2 infected patients were observed firstly by researchers in China. Thus untreated patients revealed large arterial pulmonary clots can put permanent pressure on the heart, leading to cardiac attack. Anxiety about the coronavirus outbreak is extremely common. Therefore, some individual may showed a huge concern that can disturb their regular life. Stress and fear from infection can be devastating and cause powerful excitements in children and adults. Therefore this study aims to shed light on pathophysiological consequences and stressful conditions of COVID-19 in patients including many of boby organs and ABO system.
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